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Calcium D Glucarate by Life Extension Review

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Microbes can utilize some growth factors like vitamins and this become the basis of microbial assay of vitamins. In this video there is explanation about the process how to assay calcium pantothenate using lactobacillus plantarum.
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Randy Clark, MD from Coral Desert Orthopedics in St. George Utah narrates a video demonstration of calcium removal from a rotator cuff. He is an orthopedic surgeon who has sports medicine/arthroscopic fellowship training. Dr. Clark has a particular interest in minimally invasive shoulder, hip, knee, foot and ankle tendon and ligament reconstruction.

Dr. Samin Sharma, a leading interventional cardiologist at The Mount Sinai Hospital, is the first in the world to use the newly U.S. Food and Drug Administration (FDA) approved device for the treatment of severely calcified coronary arteries before the placement of a cardiac stent to open a blocked artery. This video shows Dr. Sharma performing the first procedure in the world using the new technology inside the Cardiac Cath Lab at Mount Sinai Heart. The new device is called the Diamondback 360® Coronary Orbital Atherectomy System. It’s spinning electrically powered 1.25 mm diamond-coated crown is located on a thin cardiac catheterization guide wire and works within seconds to reduce the amount of hard calcium buildup in a coronary artery. The small calcium particles sanded from the artery’s wall are then naturally discarded from the heart and the body. The atherectomy system made by Cardiovascular Systems, Inc. was just FDA approved on Oct. 21. It is the first new coronary atherectomy system in more than two decades.
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A step-by-step description of how to write the electron configuration for Calcium (Ca).

In order to write the Ca electron configuration we first need to know the number of electrons for the Ca atom (there are 20 electrons). When we write the configuration we’ll put all 20 electrons in orbitals around the nucleus of the Calcium atom.

In this video we’ll use the electron configuration chart to help us write the notation for Calcium. Note that the last term in the Calcium electron configuration will be 1s2 2s2 2p6 3s2 3p6 4s2.

The configuration notation provides an easy way for scientists to write and communicate how electrons are arranged around the nucleus of an atom. This makes it easier to understand and predict how atoms will interact to form chemical bonds.

Get more chemistry help at http://www.thegeoexchange.org/chemistry/
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Symptoms of Not Getting Enough Calcium- Thomas DeLauer

Do you need more calcium? In this video we look at some of the ways to determine if you may need more calcium and what it does within the body and how it works with magnesium.

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References

https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC1334730/
https://www.ahajournals.org/doi/full/10.1161/jaha.116.003815
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(USMLE topics) Calcium metabolism and hypocalcemia: causes, symptoms, pathology, treatment. This video is available for instant download licensing here : https://www.alilamedicalmedia.com/-/galleries/narrated-videos-by-topics/electrolyte-acid-base-balance/-/medias/33d05c38-58ef-4d48-8ab8-654155fc3c84-hypocalcemia-narrated-animation
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In the body, most calcium is located in bones, only about 1% is in the blood and extracellular fluid. The amount of calcium in circulation is regulated by 2 hormones: parathyroid hormone, PTH, and calcitriol. When serum calcium level is low, PTH is UPregulated. PTH acts to PROMOTE calcium release from bones and REDUCE calcium loss from urine. At the same time, it stimulates production of calcitriol, which promotes absorption of calcium in the small intestine while also INcreases RE-absorption in the kidney. Together, they bring UP calcium levels back to normal. The REVERSE happens when calcium level is high. This feedback loop keeps serum calcium concentrations within the normal range.
HYPOcalcemia refers to ABnormally LOW levels of calcium in the blood and is generally defined as serum calcium level LOWER than 2.1 mmol/L. Because the total serum calcium includes albumin-bound and free-ionized calcium, of which only the LATTER is physiologically active, calcium levels must be corrected to account for albumin changes. For example, DEcreased albumin levels, such as in liver diseases, nephrotic syndrome, or malnutrition, produce LOWER serum calcium values but the amount of FREE calcium may STILL be normal. On the other hand, in conditions with high blood pH, albumin binds MORE calcium; leaving LESS FREE-ionized calcium in the serum while the total calcium level may appear normal.
The most common cause of hypocalcemia is PTH deficiency resulting from DEcreased function of the parathyroid glands, or HYPOparathyroidism. HYPOparathyroidism, in turn, may be caused by a variety of diseases and factors. These include:
– accidental removal or damage of the parathyroid glands during a surgery
– autoimmune disorders
– congenital disorders: mutations that set the “normal calcium levels” to a lower value
– other genetic disorders that produce underdeveloped or non-functional parathyroid glands
– magnesium deficiency
Other causes of hypocalcemia include low vitamin D intake/production, and excessive loss of calcium from the circulation such as in kidney diseases, tissue injuries or gastrointestinal diseases.
While chronic moderate hypocalcemia may be Asymptomatic, ACUTE and severe hypocalcemia can be life-threatening. Most symptoms of acute hypocalcemia can be attributed to the effect it has on action potential generation in neurons. Because extracellular calcium INHIBITS sodium channels, and consequently DEpolarization, REDUCED calcium level makes it EASIER for depolarization to occur. Hypocalcemia therefore INCREASES neuronal excitability, causing neuromuscular irritability and muscle spasms. Early symptoms often include numbness and tingling sensations around the mouth, in the fingers and toes. As the disease progresses, muscle spasms may manifest as tetany, wheezing, voice change, and dysphagia. Seizures may occur in severe cases. Effects of hypocalcemia on cardiac function include long QT interval due to prolonged ST fragment, congestive heart failure and hypotension.
Acute hypocalcemia should be treated promptly with intravenous calcium. Chronic hypocalcemia is usually treated with oral calcium and possibly vitamin D supplementation.
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